Journal of Ayurveda and Integrated Medical Sciences

2025 Volume 10 Number 9 September
Publisherwww.maharshicharaka.in

An Ayurvedic approach in the management of Paad Shoth w.s.r. to Foot Edema - A Case Study

Choudhary P1*, Rajoria S2, Zahir R3
DOI:10.21760/jaims.10.9.41

1* Pushpa Choudhary, Post Graduate Scholar, Dept of Kayachikitsa, Madan Mohan Malviya Government Ayurvedic College and Hospital, Udaipur, Rajasthan, India.

2 Seeta Rajoria, Associate Professor, Dept of Kayachikitsa, Madan Mohan Malviya Government Ayurvedic College and Hospital, Udaipur, Rajasthan, India.

3 Ruhi Zahir, Assistant Professor, Dept of Kayachikitsa, Madan Mohan Malviya Government Ayurvedic College and Hospital, Udaipur, Rajasthan, India.

Introduction: Paad Shoth (foot swelling) accompanied by elevated SGPT and SGOT levels is often indicative of liver dysfunction (Yakrit Vikara) and Kapha-Pitta doshic imbalance. Impaired Agni and accumulation of Ama (toxins) obstruct Srotas, leading to fluid retention and peripheral edema.

Materials and Methods: A 26-year-old male patient with elevated liver enzymes came to OPD of kayachikitsa dept. The patient complaint both lower leg swelling with pitting edema since 4-year, constipation and low appetite, fatigue, and general discomfort. Based on Ayurvedic principles, the condition was diagnosed as an imbalance in Pitta and Kapha doshas, where Pitta aggravation was linked to liver dysfunction, while Kapha contributed to fluid retention and edema. The treatment plan focused on balancing Pitta and Kapha through dietary changes, lifestyle adjustments, herbal remedies, and detoxification therapies. Punarnava, Aarogyavardhni Vati and Kutki were prescribed to reduce swelling and support liver function.

Result: After 4 weeks of treatment, the patient's swelling significantly reduced, SGPT and SGOT levels normalized, and overall vitality improved.

Conclusion: This case highlights the role of Ayurvedic principles Agni Deepana, Ama Pachana, Srotoshodhana, and Doshic balance in effectively treating Paad Shoth secondary to liver dysfunction through holistic and individualized therapy.

Keywords: elevated SGPT, elevated SGOT, liver dysfunction, Pitta imbalance, Kapha imbalance

Corresponding Author How to Cite this Article To Browse
Pushpa Choudhary, Post Graduate Scholar, Dept of Kayachikitsa, Madan Mohan Malviya Government Ayurvedic College and Hospital, Udaipur, Rajasthan, India.
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Choudhary P, Rajoria S, Zahir R, An Ayurvedic approach in the management of Paad Shoth w.s.r. to Foot Edema - A Case Study. J Ayu Int Med Sci. 2025;10(9):272-277.
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https://jaims.in/jaims/article/view/4704/

Manuscript Received Review Round 1 Review Round 2 Review Round 3 Accepted
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© 2025 by Choudhary P, Rajoria S, Zahir R and Published by Maharshi Charaka Ayurveda Organization. This is an Open Access article licensed under a Creative Commons Attribution 4.0 International License https://creativecommons.org/licenses/by/4.0/ unported [CC BY 4.0].

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Introduction

Paad Shoth or swelling of the feet, is a clinical manifestation that can range from mild puffiness to severe pitting edema. It is not a disease in itself but a symptom of an underlying pathology, commonly linked to systemic disorders involving the liver, kidneys, heart, or lymphatic system. In clinical practice, elevated liver enzymes SGPT (ALT) and SGOT (AST) are significant indicators of hepatocellular injury, pointing toward conditions such as viral hepatitis, alcoholic liver disease, non-alcoholic fatty liver disease (NAFLD), or drug-induced hepatotoxicity.[10,13]

Hepatic dysfunction compromises the synthesis of plasma proteins like albumin, leading to a reduction in plasma oncotic pressure and subsequent fluid extravasation into interstitial spaces[1], particularly in the lower extremities due to gravity. Additionally, liver dysfunction can impair portal circulation, causing portal hypertension, which further contributes to fluid accumulation in the form of ascites and peripheral edema. Renal sodium retention due to secondary hyperaldosteronism in chronic liver disease also exacerbates the problem.[2]

Ayurvedic perspective, Paad Shoth is mainly caused by an imbalance in the Kapha dosha, which is responsible for maintaining the body's structure and regulating fluid balance.[3,4,5] When the liver is involved, Pitta dosha also plays a significant role, as it governs metabolic functions and is primarily located in the liver. The disease process typically begins with Agnimandya (weakened digestive fire), which leads to the accumulation of Ama (toxic, undigested substances).

This Ama obstructs the Srotas (bodily channels), especially the Udakavaha Srotas (involved in water transport) and Rasavaha Srotas (responsible for circulating plasma). As a result, fluid distribution becomes impaired. When Rasa Dhatu (plasma tissue) is affected due to poor digestion or an unhealthy lifestyle, it can give rise to Shotha[6] (inflammation or swelling)

Clinical findings and diagnosis

The patient presented with the chief complaint of bilateral lower limb swelling, which he described as gradual in onset and progressive over the past four years.

The swelling was more pronounced in the evening hours and showed characteristics of pitting edema. The patient also reported associated symptoms including chronic constipation, with bowel movements occurring once every 2-3 days, reduced appetite, and a feeling of heaviness after meals. Additionally, he experienced persistent fatigue, general body weakness, and a sense of discomfort and lethargy, which had worsened over time and interfered with his daily activities.

The patient gave a known history of hypertension for the past 2 years, for which he was on irregular allopathic treatment. He did not report any episodes of chest pain, palpitations, dyspnea, or syncope.

He denied any history of jaundice, ascites, or known systemic illnesses such as diabetes. There was no history of prolonged medication use, alcohol intake, or exposure to hepatotoxic substances.

Laboratory investigations - The patient's underlying pathology could not be identified for the past 4 years. Later, it was diagnosed after conducting liver enzyme tests. Elevated liver enzymes SGPT and SGOT suggesting underlying hepatic dysfunction.

On Ayurvedic evaluation, the condition was assessed as a manifestation of Pitta and Kapha Dosha imbalance, wherein Pitta aggravation was linked to impaired liver function, and Kapha vitiation led to abnormal fluid retention and development of Shotha (edema) in the lower limbs.

Personal history

Name - xyz
Age - 26years
Marital status - unmarried
Occupation - student
Diet - mixed
Bowel - constipated
Appetite - Reduced
Height - 180cm
Weight - 88kg
Sleep - normal

Clinical examination

General condition: Moderate
Pulse rate: 92/minute, regular and of full volume
Heart rate: 92/minute
Blood pressure: 140/90 mmHg (Right arm, sitting)
Respiration rate: 20/minute


Ashtavidha Pariksha[7]

Naadi (Pulse): Vata Pradhana Nadi, regular

Mutra (Urine): Quantity: 900-1100 ml/day, Frequency: Normal, Color: Normal, Odor:Normal

Mala (Stool): Consistency: Hard, Color: Yellow, Frequency: Irregular (needing medication)

Jihva (Tongue): coated, Color: Normal

Shabda (Speech): Normal

Sparsha (Touch): Dry skin

Drik (Vision): Normal vision

Akriti (Body Built): Normal

Dashavidha Pariksha[3]

Prakriti (Constitution)

Sharirika (Physical): Pittakaphaja
Manasika (Mental): Rajasika

Vikriti (Disease State)

Dosha: Kapha, Pitta

Dushya (Tissue elements affected): Rasa (Plasma), Rakta (Blood), Sira (Vessels)

Adhisthana (Seat of the disease): Pad (lower leg)

Srotodushti (Channel impairment): Vimargagamana (detour)

Saara (Essence): Madhyama (Average)

Samhanana (Physique): Madhyama (Average)

Pramana (Metabolism): Madhyama (Average)

Satmya (Dietary preferences): Sarva Rasa (Can tolerate all tastes)

Satva (Mental state): Prabal (strong mental strength)

Ahara Shakti (Digestive power): Madhyama (Average)

Vyayama Shakti (Physical strength): Prabal (strong physical strength)

Vaya (Age): Vivardhamana (17-30years)

Interventions[8,9]

The treatment plan focused on balancing Pitta, Kapha and Yakrit Vikara.

SNDrugContentDoseAnupan
1.Punarnavastak KwathPunarnava, Nimba, Patola, Shunthi, Kutki, Guduchi, Devdaru, Haritaki10gmBd (before food)-
2.Kutki ChurnaKutki2gmBd (before food)Madhu
3.Aarogyavardhni VatiShuddha Parad, Shuddha Gandhak, Loh Bhasma, Abhrak Bhasma, Tamra Bhasma, Haritaki, Bahera, Amla2 tab Bd (After food)Sukashan Jala
4.Liv. 52 DSKasani, Himsra2 tab Bd (before food)Ushna Jala

Outcome

After 4 weeks of consistent treatment, patient dem-onstrated notable clinical improvement. The bilateral lower limb swelling showed marked reduction, with pitting edema almost completely resolved. Follow-up laboratory investigations revealed that previously elevated liver enzymes SGPT & SGOT had returned to within normal refer. ranges, indicating positive hepatic response to intervention. Additionally, pati-ent reported significant improvement in appetite, regular bowel movements, & substantial increase in energy levels & overall sense of well-being, suggesting restoration of Agni (digestive/metabolic function) & reduction of Ama (toxins). No adverse effects were observed during treatment period.

TestBefore treatmentAfter treatmentNormal value
SGOT43.2 U/L31 U/L0-37 U/L
SGPT70.8 U/L38.3 U/L0-40 U/L

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Before Treatment


jaime_4704_02.JPG
After Treatment

jaime_4704_03.JPG
Before Treatment

jaime_4704_04.JPG
After Treatment

Pitting edema grading[10,11,12]

GradeIndentation DepthRebound TimeDescription
Grade 1+2mm or lessRapid (immediate)Slight pitting, no visible distortion
Grade 2+4mmRebounds in 10-15 secondsDeeper pit, rebounds quickly
Grade 3+6mmRebounds in 30+ secondsNoticeably deep pit, visible swelling
Grade 4+8mm or moreRebounds in >60 secondsVery deep pit, gross swelling, skin shiny or deformed

Before - Grade 3
After - Grade 0

Discussion

The clinical management of Paad Shoth (foot edema) associated with raised hepatic enzymes reflects complex pathophysiological interaction between liver dysfunction, fluid retention, and impaired metabolism. In Ayurveda, Paad Shoth is often correlated with Shotha Roga, which may be Dushta Udaka Dhatu or Kapha-Vata predominant, with underlying derangement of Agni and Rakta-Vaha Srotas. When associated with hepatic deran-gement, it signifies Yakrit Dushti (liver dysfunction) leading to Ama formation, fluid imbalance, and Rakta-Pitta Dushti. In this context, chosen formul-ation aims at correcting liver function, enhancing Agni, reducing edema, and detoxifying Ama.[3] Punarnavastak Kwath is classical polyherbal decoc-tion known for its potent Shothahara (anti-inflammatory) and Mutrala (diuretic) properties.[5,8] It reduces localized swelling and supports renal clearance of fluid accumulation without significant electrolyte loss. Its ingredients such as Punarnava, Daruharidra, and Erandamoola help reduce peripheral edema & improve microcircu-lation. Kutki Churna (Picrorhiza kurroa) plays crucial role as hepatoprotective agent. It acts as Yakrituttejaka (liver stimulant) and Pittavirechaka (purgative), helping reduce liver enzyme levels (SGOT and SGPT). Modern research has demons-trated Kutki's anti-inflammatory, antioxidant, and hepatoregenerative actions.[9,13] Aarogyavardhini Vati is broad-spectrum Rasayana & Hepatoprotec-tive formulation. Its components, including Tamra Bhasma, Triphala, Katuki, and Shuddha Shilajit, help detoxify liver, regulate Pitta dosha, and restore liver enzyme balance. It also supports metabolism of fats and proteins, reducing hepatic load.[8,9]


Liv 52 DS, a proprietary herbal formulation, is widely researched for its efficacy in improving liver function. It stabilizes hepatic cell membranes, enhances antioxidant defence, and promotes hepatocyte regeneration.[14] In the observed clinical response, a notable reduction in pedal edema and a gradual normalization of liver enzymes indicate the efficacy of this integrative herbal regimen. No adverse effects were noted, supporting the safety profile of the selected drugs when used under supervision. Thus, this multidrug Ayurvedic approach shows promising results in managing Paad Shoth secondary to liver dysfunction, validating the ancient principle of Samprapti Vighatana (breaking the pathogenesis) through multi-targeted therapy.[8,14]

Conclusion

This case highlights the efficacy of Ayurvedic therapeutic approaches in the management of Paad Shoth (pedal edema) associated with underlying liver dysfunction. The integration of detoxification therapies (Shodhana) and herbal formulations (Shamana) provided symptomatic relief and addressed the root cause of the condition in accordance with Ayurvedic principles. Central to the treatment was the correction of Doshic imbalance, particularly of Kapha and Pitta, enhancement of Agni (digestive/metabolic fire), and elimination of Ama (toxic waste). The use of Shothahara and Yakrituttejaka herbs such as Punarnava, Kutki and Aarogyavrdhni Vati played a pivotal role in reducing inflammation, supporting liver function, and promoting fluid drainage. Additionally, dietary modifications, lifestyle regulation, and individualized herbal interventions contributed to the overall improvement. This holistic approach reinforces the significance of Ayurveda in managing complex systemic conditions and emphasizes the need for treating not just the symptoms, but the underlying imbalances for long-term health restoration.

References

1. Hall JE. Guyton and Hall Textbook of Medical Physiology. 14th ed. Amsterdam: Elsevier; 2021. [Crossref][PubMed][Google Scholar]

2. Ralston S, Penman I, Strachan M, Hobson R. Oxford Handbook of Clinical Medicine. 10th ed. Oxford: Oxford University Press; 2022. [Crossref][PubMed][Google Scholar]

3. Agnivesha. Charaka Samhita. Vimana Sthana, Chapter 8; Chikitsa Sthana, Chapter 12/98–104. Edited by Yadavji Trikamji Acharya. Varanasi: Chaukhambha Sanskrit Sansthan; 2021. Reprint [Crossref][PubMed][Google Scholar]

4. Vagbhata. Ashtanga Hridaya. Nidana Sthana, Chapter 12. Edited by Harishastri Paradkar Vaidya. Varanasi: Chaukhambha Orientalia; 2022. Reprint [Crossref][PubMed][Google Scholar]

5. Chunekar KC, editor. Bhava Prakasha Nighantu. Madhyama Khanda – Shotha Chikitsa. Varanasi: Chaukhambha Bharati Academy; 2020. Reprint [Crossref][PubMed][Google Scholar]

6. Sushruta. Sushruta Samhita. Nidana Sthana, Chapter 13. Edited by Anant Ram Sharma. Varanasi: Chaukhambha Surbharati Prakashan; 2020. Reprint [Crossref][PubMed][Google Scholar]

7. Tripathi I, editor. Yogaratnakara. Shotha Chikitsa. Varanasi: Chaukhambha Krishnadas Academy; 2019. Reprint [Crossref][PubMed][Google Scholar]

8. Mishra S, editor. Bhaishajya Ratnavali. Shotha Rogadhikara. Varanasi: Chaukhambha Surbharati Prakashan; 2021. Reprint [Crossref][PubMed][Google Scholar]

9. Sharma S. Rasa Tarangini. Taranga 24. Edited by Kashinath Shastri. Delhi: Motilal Banarsidass; 2018. Reprint [Crossref][PubMed][Google Scholar]

10. Bickley LS. Bates’ Guide to Physical Examination and History Taking. 13th ed. Philadelphia: Wolters Kluwer; 2020. [Crossref][PubMed][Google Scholar]

11. Kumar P, Clark M. Kumar and Clark’s Clinical Medicine. 9th ed. Amsterdam: Elsevier; 2016. [Crossref][PubMed][Google Scholar]

12. Longmore M, Wilkinson I, Baldwin A, Wallin E. Oxford Handbook of Clinical Examination and Practical Skills. 2nd ed. Oxford: Oxford University Press; 2014. [Crossref][PubMed][Google Scholar]

13. World Health Organization. WHO Monographs on Selected Medicinal Plants. Vol. 2. Geneva: WHO Press; 2004. (Picrorhiza kurroa, Boerhavia diffusa) [Crossref][PubMed][Google Scholar]

14. API Textbook of Medicine. 10th ed. Mumbai: Association of Physicians of India; 2015. . [Crossref][PubMed][Google Scholar]


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